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Aging affects nearly every system in the body, from how cells replicate to how well the immune system functions

Our results from experiments using siRNA-mediated knockdowns of STAT3 and GIV suggest that the effects of IL-17 were mediated by activating STAT3/GIV signaling in NSCLC cells and subsequently up-regulating its downstream target VEGF
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The method was then applied to plasma from mice cutaneously exposed to CEES

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