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2.3 Lung fibroblasts and smooth muscle cells Small airway illness results from ongoing airway remodeling, marked by chronic mucus hypersecretion and fibroblast activation, leading to increased synthesis of extracellular matrix (ECM) proteins and subsequent hardening of the airway wall [10]

The oligomerization of PRDX2 is inhibited by ROS-induced hyperoxidation to sulfinic acid at cysteine residue

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