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Patients with hepatic steatosis are at risk of serious complications, including progression to steato hepatitis, fibrosis, cirrhosis, liver failure and hepato cellular carcinoma 4,5
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Summary Atherosclerotic lesions initiate with endothelial cell dysfunction causing modification of apoB containing lipoproteins (LDL, VLDL, remnants) and infiltration of immune cells, particularly monocytes, into the subendothelial space (Figure 1)
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