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Progress in Neuro-Psychopharmacology & Biological Psychiatry

A possible explanation of the divergent phenotypes of the TR- and deiodinase-KO mice is that, in TRKO mice, the global loss of TRs can affect epidermal homeostasis starting from embryonic development, thereby reducing the TH signal in a time window in which the TH is essential for the development of several target tissues, included the epidermis

Because of this mismatch, inflammation, and bacterial growth can manifest

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Thereby, there was the curing of the skin [53,55,103,104,105], nerve [106], tendon [50,51,107,108,109,110,111], muscle [110,111,112,113,114,115], ligament [116], and bone [117,118,119] injuries that spontaneously might not heal

Schurr and Gozal demonstrated that during neuronal activation, NADHproduced via mLDHacts as an endogenous ROS scavenger, neutralizing oxidative stress induced by glutamate excitotoxicity and thereby conferring neuroprotection (Schurr and Gozal, 2011)