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Overall, this results in increased oxidative stress and decreased glutathione-based antioxidant capacity in these mice [40]

a specific increase of fibroblast-specific GPX3 expression or a spatial shift from apically secreted soluble GPX3 to interstitial matrix-associated GPX3, reminiscent of what has been observed in a mouse model of influenza pneumonia 33

[DOI] [PMC free article] [PubMed] [Google Scholar] 160.Giudice A, Arra C, Turco MC

Furthermore, the dysfunction of ATP synthase on the inner mitochondrial membrane is frequently accompanied by loss of mitochondrial membrane potential (MMP), decreased ATP levels, and consequent electron leakage and increased ROS production (317)

These ROS injure sinusoidal endothelial cells (SECs) and hepatocytes directly, while activating NF-B and other pro-inflammatory pathways