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The answer may be poor sleep, slow recovery, low appetite, low hormone signaling, soft tissue pain, metabolic dysfunction, or inconsistent training

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The mechanism of doxorubicin actions involves an anticancer effect by intercalation into the DNA to inhibit DNA replication and transcription, and the inhibition of topoisomerase II, an enzyme important for DNA replication

However, even in one type of cancers, e.g., NSCLC, the impact of c-MET copy number on prognosis is a histological subtype dependent: c-MET amplification with a poorer prognosis in patients with adenocarcinoma but not in those with squamous cell carcinoma [21, 22]