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[DOI] [PMC free article] [PubMed] [Google Scholar] 204.Motyl K, McCabe LR

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42,83,84 The damage to the intestinal epithelial barrier and imbalance in the bacterial ecosystem in UC lead to abnormal activation of Toll-like receptor 4 (TLR4), which ultimately results in the abnormal activation and translocation of nuclear factor kappa B (NF-B)

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In contrast, SLC7A11 expression is higher in IDH-wild-type gliomas, making them more resistant to ferroptosis and promoting malignant tumor progression [2]

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