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As a tripeptide composed of glutamate, cysteine, and glycine, it functions as the primary thiol-based redox buffer, neutralizing reactive oxygen species (ROS), supporting Phase II detoxification pathways, and facilitating the excretion of xenobiotics and heavy metals [1] [8]

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doi: 10.1073/pnas.1009700107

The results indicated that N-terminal modifications were particularly effective at enhancing metabolic stability, whereas C-terminal amidation offered more modest protection

NO system dependence of atropine-induced mydriasis and L-NAME- and L-arginine-induced miosis: reversal by the pentadecapeptide BPC 157 in rats and guinea pigs