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Two bioinformatics analyses identified three (ACO1, HCRA1, and MMD) and six (CYBB, CTSB, SLC38A1, TLR4, PEX2, and ABCC1) FRGs, respectively, with strong diagnostic specificity in the orbital fibroblasts of thyroid-associated orbitopathy (TAO) patients (63, 64)

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Their mechanism involves restoring the acetylation of -tubulin in neurons, which stabilizes microtubules and enhances mitochondrial transport 63

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others include cardiovascular injury, causing heart attacks and heart failure