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ABCC1 may be relatively more important in terminal detoxification in nonpolarized cell types (Nies and Keppler, 2007)
In another study, metabolome analysis of 110 metabolites in CD44v9-positive and CD44v9-negative tumors suggested that CD44v9 could increase pentose phosphate pathway flux and sustain glutathione expression in gastric cancer cells [121]
This laboratory-grade compound is a synthetic peptide derivative designed to mimic and amplify the activity of hepatocyte growth factor (HGF), a key player in neuronal growth and repair
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The elevation of muscle Chac1 expression is associated with reduced glutathione level
