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Recent advancements in quercetin derivatives, such as quercetin1,2,3triazole hybrids, have shown enhanced cholinesterase inhibition and antioxidant activity, surpassing standard drugs like galantamine, thus highlighting quercetin's potential in multitarget AD therapy (Carreiro et al

Gleeson M

Studies have suggested that increased susceptibility to KBD in selenium-deficient populations might result from a reduced antioxidant protection associated with polymorphisms in GPX genes (60, 61), but polymorphisms in other genes have also been implicated (62)

On the test day, mice were allowed to freely explore chambers for 15 minutes without injections

Such neurotransmitter generalization can be in a wide therapy range previously seen in terms of cytoprotection and evidenced implementation with the pleiotropic agents beneficial activity [1]

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