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Description
Reduced mitochondrial number and their increased deformation may damage the normal oxidative decomposition of fat, thereby increasing hepatic fat content

Lewis, Frank Greenway, Tori R

J Hazard Mater 430:e128476

[Crossref] Shiraki M, Shimizu M, Moriwaki H, Okita K, Koike K (2017) Carnitine dynamics and their effects on hyperammonemia in cirrhotic Japanese patients

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10.1073/pnas.1316569111 Summary Keywords trimethylamine N-oxide, chronic kidney disease, gut microbiota, targeted TMAO drugs, mechanism, treatment Citation Zixin Y, Lulu C, Xiangchang Z, Qing F, Binjie Z, Chunyang L, Tai R and Dongsheng O (2022) TMAO as a potential biomarker and therapeutic target for chronic kidney disease: A review