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Description
3.1 Inflammatory respiratory diseases NSPs are released in the airways of patients with multiple types of chronic pulmonary diseases such as COPD, AATD, bronchiectasis, and CF, and have been implicated as key mediators of chronic inflammation and disease progression (59)
These changes suggest that GCH1 overexpression does not protect all PUFA-PLs, but selectively blocks the oxidation of specific PUFA-PLs
doi:10.3390/ijms23062948 324
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Diagnostics 14 , 2229 (2012)
sporogenes , which produced more isobutanoic than butanoic acid
