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In pancreatic ductal adenocarcinoma (PDAC), tumor-intrinsic factors, notably KRAS mutation status, and iatrogenic influences like chemotherapy-induced dysbiosis, dramatically reshape BA profiles and modulate the local TIME (160)

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These proteins belong to the (nicotinamide adenine dinucleotide) NAD+ dependent class III histone deacetylase (HDAC) family of enzymes and different types are present in different compartments of cells such as cytoplasm, mitochondria, and nucleus

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