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Description
This explains why, in patients with mutations in the SLC6A8 gene, there is a lack of sufficient brain creatine
In Adipoq / mice, P5 treatment can achieve similar effects
Endothelial Ca+-activated K+ channels in normal and impaired EDHF-dilator responses-relevance to cardiovascular pathologies and drug discovery
Disruption of Dhcr7 and Insig1/2 in cholesterol metabolism causes defects in bone formation and homeostasis through primary cilium formation
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Depression can also arise from post-synaptic processes and from feedback activation of presynaptic receptors
