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Given their potential to regulate multiple pathways and genes, (-)-Vestitol and Salviolone may exert beneficial or unforeseen effects on various physiological systems over extended periods

International Journal of Pharmaceutics, 493 (1-2), 460-465

Some authors reported that there are additional mechanisms in MEM neuroprotection like nerve growth factor upregulation that activates the tropomyosin receptor kinase A (Trk A) signaling, which eventually inhibits p75 neurotrophin receptor (p75NTR), molecules implicated in neuronal plasticity [48]

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