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Eustis et al

C57BL/6 in experiment 1, NIH-Swiss in experiment 2) infected with 60 S

Dysfunction of CI is a defining feature of circadian disruption and diet-induced obesity in adipose tissue, linking clock misalignment to visceral adipocyte hypertrophy, ectopic lipid deposition in liver and impaired glucose tolerance

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After TBI, mitochondrial membrane disruption not only critically impairs oxidative phosphorylation at the site of electron transport but also activates downstream signaling pathways (ROS, caspases, calpains, etc.) that lead to cell death [76]

The ratio of GSH to GSSG determines cell redox status