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Treatment of SW480 and HCT116 cells with Andrographis significantly reduced cell viability as well as GPX4 and FSP1 expression

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Taken together, hyperhomocysteinemia triggers the development and progression of PD by different mechanisms, including oxidative stress, mitochondrial dysfunction, apoptosis, and endothelial dysfunction [Fig

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mRNA levels were examined by quantitative RT-PCR and normalized to -actin mRNA expression